Article Project 2 Project Research on the Elucidation of Generating Mechanism of Damaged Protein Induced by Aging and Irradiation (28P2)
I. Annual Summary of Experimental Research Activities. I-1. PROJECT RESEARCHES

Fujii, N.

2016 ( APRIL 2016 - MARCH 2017 )  , pp.3 - 10 , 2017-07 , The Research Reactor Institute, Kyoto University
ISSN:0919-1038
NCID:AA10912308
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In case that corrections are made, an errata will be provided in the following webpage: http://www.rri.kyoto-u.ac.jp/PUB/report/PR/ProgRep2016/ProgRep2016.html
PR2-1 Consideration for the Distribution of D-Aspartyl Endopeptidases Activity in Various Living Things /T. Kinouchi and N. Fujii (28P2-1) [4]
PR2-2 Damage to Biological Molecules Induced by Ionizing Irradiation and Biological Defense Mechanisms against Ionizing Radiation III /T. Saito and N. Fujii (28P2-2) [5]
PR2-3 Separation Condition of the Prion Peptide (106-126) after Treatment of Protein L-Isoaspartyl Methyltransferase (PIMT) /Y. Sadakane and N. Fujii (28P2-3) [6]
PR2-4 Analysis of Hearing Impairments in Mice Exposed to Environmental Stress /N. Ohgami and N. Fujii (28P2-4) [7]
PR2-5 Identification of Contiguous β-aspratyl Residues in Peptide Using MS /N. Fujii et al. (28P2-5) [8]
PR2-6 The Stability of D-β-Asp :kinetics of the Competitive Reactions of Isomerization and Peptide bond Cleavage /K. Aki et al. (28P2-6) [9]
PR2-7 Change of Protein Function by the Replacement of a Single Aspartyl Isomer in a Protein /N. Fujii et al. (28P2-7) [10]
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